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A lesion in the SCN eliminates the glucocorticoid circadian rhythm (162)
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Previous genetic studies have shown that conditional knockout of Ctnnb1 in the skin impedes the transition of hair follicles from the resting phase to the growth phase, leading to arrested hair growth ( Ctnnb1 is significantly upregulated in anwuligan-treated HFSCs, but this effect was not observed in skin tissues
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